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Neurocritical Care and Emergencies

Manual: General · Subject: Neurology

Apply first-principles thinking to coma, increased intracranial pressure, spinal cord compression, and acute weakness.

Emergency Neurology Mindset

Core priorities

In neurologic emergencies, the first question is not the diagnosis but whether the patient is protecting airway, breathing, and circulation while preserving brain and spinal cord function.

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Time-sensitive approach

Early recognition and escalation matter because neurologic injury can become irreversible quickly.

Common emergencies

Coma

  • Evaluate brainstem reflexes, glucose, toxic-metabolic causes

Raised intracranial pressure

  • Headache, vomiting, papilledema, declining consciousness

Which bedside finding most strongly suggests brainstem function is impaired in a comatose patient?

Why is point-of-care glucose checked early in altered mental status?

Raised Intracranial Pressure and Herniation

Danger signs

Increasing intracranial pressure can compress brain structures and cause herniation syndromes. Signs include progressive somnolence, vomiting, pupillary changes, and abnormal posturing.

Initial response to suspected raised ICP

  1. 1

    Step 1: Call for urgent help and monitor airway, breathing, and circulation.

  2. 2

    Step 2: Elevate the head and optimize oxygenation and ventilation.

  3. 3

    Step 3: Rapidly identify mass lesion, hydrocephalus, or hemorrhage.

  4. 4

    Step 4: Escalate to definitive neurocritical or neurosurgical management.

A dilated, poorly reactive pupil in a deteriorating patient can be a sign of:

What symptom triad should make you worry about spinal cord compression?

Acute Weakness Triage

Potential causes of acute weakness

CategoryExamples
Central nervous systemStroke, cord lesion
Peripheral nerve/rootGuillain-Barré syndrome, radiculopathy
Neuromuscular junctionMyasthenic crisis, botulism
MuscleInflammatory or metabolic myopathy

Ascending weakness with areflexia after a recent infection suggests:

Why is respiratory monitoring important in acute neuromuscular weakness?